NURS 6501 Acute Lymphoblastic Leukemia
Walden University NURS 6501 Acute Lymphoblastic Leukemia-Step-By-Step Guide
This guide will demonstrate how to complete the Walden University NURS 6501 Acute Lymphoblastic Leukemia assignment based on general principles of academic writing. Here, we will show you the A, B, Cs of completing an academic paper, irrespective of the instructions. After guiding you through what to do, the guide will leave one or two sample essays at the end to highlight the various sections discussed below.
How to Research and Prepare for NURS 6501 Acute Lymphoblastic Leukemia
Whether one passes or fails an academic assignment such as the Walden University NURS 6501 Acute Lymphoblastic Leukemia depends on the preparation done beforehand. The first thing to do once you receive an assignment is to quickly skim through the requirements. Once that is done, start going through the instructions one by one to clearly understand what the instructor wants. The most important thing here is to understand the required format—whether it is APA, MLA, Chicago, etc.
After understanding the requirements of the paper, the next phase is to gather relevant materials. The first place to start the research process is the weekly resources. Go through the resources provided in the instructions to determine which ones fit the assignment. After reviewing the provided resources, use the university library to search for additional resources. After gathering sufficient and necessary resources, you are now ready to start drafting your paper.
How to Write the Introduction for NURS 6501 Acute Lymphoblastic Leukemia
The introduction for the Walden University NURS 6501 Acute Lymphoblastic Leukemia is where you tell the instructor what your paper will encompass. In three to four statements, highlight the important points that will form the basis of your paper. Here, you can include statistics to show the importance of the topic you will be discussing. At the end of the introduction, write a clear purpose statement outlining what exactly will be contained in the paper. This statement will start with “The purpose of this paper…” and then proceed to outline the various sections of the instructions.
How to Write the Body for NURS 6501 Acute Lymphoblastic Leukemia
After the introduction, move into the main part of the NURS 6501 Acute Lymphoblastic Leukemia assignment, which is the body. Given that the paper you will be writing is not experimental, the way you organize the headings and subheadings of your paper is critically important. In some cases, you might have to use more subheadings to properly organize the assignment. The organization will depend on the rubric provided. Carefully examine the rubric, as it will contain all the detailed requirements of the assignment. Sometimes, the rubric will have information that the normal instructions lack.
Another important factor to consider at this point is how to do citations. In-text citations are fundamental as they support the arguments and points you make in the paper. At this point, the resources gathered at the beginning will come in handy. Integrating the ideas of the authors with your own will ensure that you produce a comprehensive paper. Also, follow the given citation format. In most cases, APA 7 is the preferred format for nursing assignments.
How to Write the Conclusion for NURS 6501 Acute Lymphoblastic Leukemia
After completing the main sections, write the conclusion of your paper. The conclusion is a summary of the main points you made in your paper. However, you need to rewrite the points and not simply copy and paste them. By restating the points from each subheading, you will provide a nuanced overview of the assignment to the reader.
How to Format the References List for NURS 6501 Acute Lymphoblastic Leukemia
The very last part of your paper involves listing the sources used in your paper. These sources should be listed in alphabetical order and double-spaced. Additionally, use a hanging indent for each source that appears in this list. Lastly, only the sources cited within the body of the paper should appear here.
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Sample Answer for NURS 6501 Acute Lymphoblastic Leukemia
Scenario 1: Acute Lymphoblastic Leukemia (ALL)An 11-year-old boy is brought to the clinic by his parents who states that the boy has not been eating and listless. The mother also notes that he has been easily bruising without trauma as he says he is too tired to go out and play. He says his bones hurt sometimes. Mother states the child has had intermittent fevers that respond to acetaminophen. Maternal history negative for pre, intra, or post-partum problems. PMH: Negative. Easily reached developmental milestones. PE: reveals a thin, very pale child who has bruises on his arms and legs in no particular pattern. LABS: CBC revealed Hemoglobin of 6.9/dl, hematocrit of 19%, and platelet count of 80,000/mm3. The CMP demonstrated a blood urea nitrogen (BUN) of 34m g/dl and creatinine of 2.9 mg/dl. DIAGNOSIS: acute leukemia and renal failure and immediately refers the patient to the Emergency Room where a pediatric hematologist has been consulted and is waiting for the boy and his parents. CONFIRMED DX: acute lymphoblastic leukemia (ALL) was made after extensive testing. Question1. Explain what ALL is? |
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Pediatric disorders can present unique challenges to patients, families, and healthcare providers. Disorders in these areas are complicated by the fact that young patients can have difficulties communicating symptoms. Furthermore, the manner in which disease and disorders manifest in children may be unique.
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APRNs working to support these patients and their loved ones must demonstrate not only support and compassion, but expertise to communicate and guide understanding of diagnoses and treatment plans. This includes an understanding of disease and disorders at the pediatric level.
This week, you examine pathophysiology in pediatrics. You apply key terms, concepts, and principles in this area to demonstrate an understanding of the impact they have on altered physiology in children.
Learning Objectives NURS 6501 Knowledge Check Pediatrics
Students will:
Analyze concepts and principles of pathophysiology across the lifespan
Also Read:
NURS 6501 Immune Thrombocytopenia Purpura (ITP) Pathophysiology
Learning Resources
Required Readings (click to expand/reduce)
McCance, K. L. & Huether, S. E. (2019). Pathophysiology: The biologic basis for disease in adults and children (8th ed.). St. Louis, MO: Mosby/Elsevier.
Chapter 14: Cancer in Children, including Summary Review
Chapter 20: Alterations of Neurologic Function in Children (stop at Childhood tumors); Summary Review
Chapter 34: Alterations of Cardiovascular Function in Children (stop at Defects
decreasing pulmonary blood flow); Summary Review
Chapter 37: Alterations of Pulmonary Function in Children (stop at Congenital malformations); Summary Review
Chapter 40: Alterations of Renal and Urinary Tract Function in Children, including Summary Review
Chapter 43: Alterations of Digestive Function in Children, including Summary Review
Chapter 46: Alterations of Musculoskeletal Function in Children (stop at Avascular diseases); (start at Cerebral palsy) (musculoskeletal tumors in children); Summary Review
Chapter 48: Alterations of the Integument in Children, including Summary Review
NURS 6501 Knowledge Check Pediatrics Chapter 50: Shock, Multiple Organ Dysfunction Syndrome, and Burns in Children, including Summary Review
U.S. National Library of Medicine. (2019). Normal growth and development. Retrieved from https://medlineplus.gov/ency/article/002456.htm
Document: NURS 6501 Final Exam Review (PDF document)
Note: Use this document to help you as you review for your Final Exam in Week 11.
Required Media (click to expand/reduce)
Knowledge Check: Pediatrics
In this exercise, you will complete a 10- to 20-essay type question Knowledge Check to gauge your understanding of this module’s content.
Possible topics covered in this Knowledge Check include:
Growth and development
Normal growth patterns
Scoliosis (ortho)
Kawasaki
Alterations in children
Congenital (heart syndrome)
PDAs
Sudden Infant Death Syndrome (SIDS)
Asthma
Lead poisoning and effects on neurological functioning
Sickle cell
Hemophilia
Photo Credit: laflor / E+ / Getty Images
(Note: It is strongly recommended that you take the Knowledge Check at least 48 hours before taking the Final Exam.)
Complete the Knowledge Check By Day 5 of Week 11
To complete this Knowledge Check:
Module 8 Knowledge Check
Final Exam
This 101-question exam is a test of your knowledge in preparation for your certification exam. No outside resources, including books, notes, websites, or any other type of resource, are to be used to complete this exam. You are expected to comply with Walden University’s Code of Conduct.
This exam will be on topics covered in Weeks 7, 8, 9, 10, and 11. Prior to starting the exam, you should review all of your materials. This exam is timed with a limit of 2 hours for completion. When time is up, your exam will automatically submit.
(Note: It is strongly recommended that you take the Knowledge Check at least 48 hours before taking the Final Exam.)
Photo Credit: Getty Images
By Day 7 of Week 11
Complete and submit your Final Exam.
To complete your exam:
Final Exam
What’s Coming Up?
Congratulations! After you have finished all of the assignments for this week, you have completed the course. Please submit your Course Evaluation by Day 7.
Results Displayed All Answers, Submitted Answers, Correct Answers, Feedback, Incorrectly Answered Questions
Question 1
1 out of 1 points
Correct
An adolescent that uses anabolic steroids to increase muscle mass is at risk for developing which type of cancer?
Selected Answer:
Correct
Hepatocellular carcinoma
Answers:
Neuroblastoma
Acute myelogenous leukemia
Vaginal adenocarcinoma
Correct
Hepatocellular carcinoma
Question 2
1 out of 1 points
Correct
Which of the following about Roseaola is FALSE?
Selected Answer:
Correct
severely pruritic
Answers:
is also known as exanthema subitum
Correct
severely pruritic
rash appears after high fever
most common in infants
Question 3
1 out of 1 points
Correct
Children who have inherited deficiencies of antithrombin III or proteins C or S have increased risk for _______________
Selected Answer:
Correct
Thrombosis
Answers:
Hemorrhage
Sickle cell anemia
Correct
Thrombosis
Anemia
Question 4
0 out of 1 points
Incorrect
Which of the following characteristics of Impetigo is FALSE?
Selected Answer:
Incorrect
Commonly caused by staphylococci
Answers:
Correct
Is mildly contagious
Commonly caused by staphylococci
class lesions are honey colored crusts
outbreaks usually begin with vesicles
Question 5
1 out of 1 points
Correct
A child with Fanconi anemia is at risk for developing which type of cancer?
Selected Answer:
Correct
Acute myelogenous leukemia
Answers:
Neuroblastoma
Correct
Acute myelogenous leukemia
Vaginal adenocarcinoma
Hepatocellular carcinoma
Question 6
1 out of 1 points
Correct
Children who have atopic dermatitis often have elevated levels of ______________________.
Selected Answer:
Correct
IgE
Answers:
Correct
IgE
IgM
IgG
IgN
Question 7
1 out of 1 points
Correct
What is the function of G6PD?
Selected Answer:
Correct
It is a normal enzyme that protects erythrocytes from oxidative damage
Answers:
It is an enzyme that causes the breakdown of red blood cells
Correct
It is a normal enzyme that protects erythrocytes from oxidative damage
It is an enzyme that causes the formation of red blood cells
It is an enzyme that inhibits coagulation
Question 8
1 out of 1 points
Correct
During the first five years of life, the respiratory system continues to develop and the number of what structure increases?
Selected Answer:
Correct
alveoli
Answers:
conducting airways
Correct
alveoli
bronchi
bronchioles
Question 9
1 out of 1 points
Correct
Red, round, and scaling patches with a clear center that occur mostly on the face, trunk, and limbs in an asymmetrical distribution describes which of the following conditions?
Selected Answer:
Correct
Tinea corporis
Answers:
Atopic dermatitis
Correct
Tinea corporis
Impetigo
Staphylococcal scalded-skin syndrome
Question 10
1 out of 1 points
Correct
Rotavirus is the leading cause of what symptom in infants and young children?
Selected Answer:
Correct
acute diarrhea
Answers:
Correct
acute diarrhea
necrotizing enterocolitis
constipation
abdominal pain
Question 11
1 out of 1 points
Correct
A ventricular septal defect will cause what type of blood shunting?
Selected Answer:
Correct
Left to right
Answers:
Correct
Left to right
Right to left
No shunt
Question 12
1 out of 1 points
Correct
Respiratory distress syndrome in the newborn is caused by the deficiency of what substance?
Selected Answer:
Correct
surfactant
Answers:
fibrin
hemoglobin
Correct
surfactant
iron
Question 13
1 out of 1 points
Correct
The most common cause of chronic hepatitis in children is:
Selected Answer:
Correct
B and C
Answers:
A and B
A and C
Correct
B and C
B and D
Question 14
1 out of 1 points
Correct
Von Willebrand disease is a genetic condition that causes: (thrombotic, hemorrhagic) condition.
Selected Answer:
Correct
hemorrhage
Answers:
thrombosis
Correct
hemorrhage
anemia
infection
Question 15
1 out of 1 points
Correct
What medication should a child with G6PD deficiency avoid because it can trigger a hemorrhage?
Selected Answer:
Correct
aspirin
Answers:
Vitamin D
iron
penicillin
Correct
aspirin
Question 16
1 out of 1 points
Correct
Pruritic linear lesions that itch more at night that may have more vesicles and papules describes which of the following conditions?
Selected Answer:
Correct
Scabies
Answers:
Atopic dermatitis
Tinea corporis
Impetigo
Correct
Scabies
Question 17
1 out of 1 points
Correct
Children with Down’s Syndrome has an increased risk of developing which type of cancer?
Selected Answer:
Correct
Leukemia
Answers:
Retinoblastoma
Correct
Leukemia
Wilms tumor
Neuroblastoma
Question 18
1 out of 1 points
Correct
Which of the following is true of people that have sickle cell anemia?
Selected Answer:
Correct
People who have sickle cell anemia have two copies of the mutated gene and produce only hemoglobin S
Answers:
Correct
People who have sickle cell anemia have two copies of the mutated gene and produce only hemoglobin S
People who have sickle cell anemia have one mutated gene and produce a mixture of hemoglobin A and hemoglobin S
People who have sickle cell anemia have one copy of the mutated gene and produce only hemoglobin A
People who have sickle cell anemia have two copies of the mutated gene and produce a mixture of hemoglobin A and hemoglobin S
Question 19
1 out of 1 points
Correct
The development of Kawasaki disease in young children is caused by:
Selected Answer:
Correct
vasculitis of unknown cause
Answers:
Correct
vasculitis of unknown cause
Epstein Barr Virus
Genetic malformation
Medication interation
Question 20
1 out of 1 points
Correct
Cerebral palsy involves what type of defects?
Selected Answer:
Correct
motor
Answers:
Correct
motor
sensory
cognitive
developmental
Sample Answer 2 for NURS 6501 Acute Lymphoblastic Leukemia
Question 4
4 out of 4 points
Scenario 2: Sickle Cell Disease (SCD)A 15-year-old male with known sickle cell disease (SCD) present to the ER in sickle cell crisis. The patient is crying with pain and states this is the third acute episode he has had in the last 10-months. Both parents are present and appear very anxious and teary eyed. A diagnosis of acute sickle cell crisis was made. Question 1. Discuss the genetic basis for SCD. |
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· Question 5
4 out of 4 points
Scenario 3: Hemophilia8-month infant is brought into the office due to a swollen right knee and excessive bruising. The parents have noticed bruising about a month ago but thought the bruising was due to the attempts to crawl. They became concerned when the baby woke up with a swollen knee. Infant up to date on all immunizations, has not had any medical problems since birth and has met all developmental milestones. FH: negative for any history of bleeding disorders or other major genetic diseases. PE: within normal limits except for obvious bruising on the extremities and right knee. Knee is swollen but no warmth appreciated. Range of motion of knee limited due to the swelling. DIAGNOSIS: hemophilia A. Question 1. What is the pathophysiology of Hemophilia |
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