NURS 6501 Concepts of Neurological and Musculoskeletal Disorders – Part 1
Walden University NURS 6501 Concepts of Neurological and Musculoskeletal Disorders – Part 1-Step-By-Step Guide
This guide will demonstrate how to complete the Walden University NURS 6501 Concepts of Neurological and Musculoskeletal Disorders – Part 1 assignment based on general principles of academic writing. Here, we will show you the A, B, Cs of completing an academic paper, irrespective of the instructions. After guiding you through what to do, the guide will leave one or two sample essays at the end to highlight the various sections discussed below.
How to Research and Prepare for NURS 6501 Concepts of Neurological and Musculoskeletal Disorders – Part 1
Whether one passes or fails an academic assignment such as the Walden University NURS 6501 Concepts of Neurological and Musculoskeletal Disorders – Part 1 depends on the preparation done beforehand. The first thing to do once you receive an assignment is to quickly skim through the requirements. Once that is done, start going through the instructions one by one to clearly understand what the instructor wants. The most important thing here is to understand the required format—whether it is APA, MLA, Chicago, etc.
After understanding the requirements of the paper, the next phase is to gather relevant materials. The first place to start the research process is the weekly resources. Go through the resources provided in the instructions to determine which ones fit the assignment. After reviewing the provided resources, use the university library to search for additional resources. After gathering sufficient and necessary resources, you are now ready to start drafting your paper.
How to Write the Introduction for NURS 6501 Concepts of Neurological and Musculoskeletal Disorders – Part 1
The introduction for the Walden University NURS 6501 Concepts of Neurological and Musculoskeletal Disorders – Part 1 is where you tell the instructor what your paper will encompass. In three to four statements, highlight the important points that will form the basis of your paper. Here, you can include statistics to show the importance of the topic you will be discussing. At the end of the introduction, write a clear purpose statement outlining what exactly will be contained in the paper. This statement will start with “The purpose of this paper…” and then proceed to outline the various sections of the instructions.
How to Write the Body for NURS 6501 Concepts of Neurological and Musculoskeletal Disorders – Part 1
After the introduction, move into the main part of the NURS 6501 Concepts of Neurological and Musculoskeletal Disorders – Part 1 assignment, which is the body. Given that the paper you will be writing is not experimental, the way you organize the headings and subheadings of your paper is critically important. In some cases, you might have to use more subheadings to properly organize the assignment. The organization will depend on the rubric provided. Carefully examine the rubric, as it will contain all the detailed requirements of the assignment. Sometimes, the rubric will have information that the normal instructions lack.
Another important factor to consider at this point is how to do citations. In-text citations are fundamental as they support the arguments and points you make in the paper. At this point, the resources gathered at the beginning will come in handy. Integrating the ideas of the authors with your own will ensure that you produce a comprehensive paper. Also, follow the given citation format. In most cases, APA 7 is the preferred format for nursing assignments.
How to Write the Conclusion for NURS 6501 Concepts of Neurological and Musculoskeletal Disorders – Part 1
After completing the main sections, write the conclusion of your paper. The conclusion is a summary of the main points you made in your paper. However, you need to rewrite the points and not simply copy and paste them. By restating the points from each subheading, you will provide a nuanced overview of the assignment to the reader.
How to Format the References List for NURS 6501 Concepts of Neurological and Musculoskeletal Disorders – Part 1
The very last part of your paper involves listing the sources used in your paper. These sources should be listed in alphabetical order and double-spaced. Additionally, use a hanging indent for each source that appears in this list. Lastly, only the sources cited within the body of the paper should appear here.
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Sample Answer for NURS 6501 Concepts of Neurological and Musculoskeletal Disorders – Part 1
It is no coincidence that the neurological system and the musculoskeletal system share both anatomical, physiological, and functional connectivity. This structure-function relationship between the neuronal and muscular topology is responsible for influencing the disease process, the pathogenesis, presentation, diagnostic formulation, and treatment of both systems. Therefore, an understanding of the symptoms of alterations in neurological and musculoskeletal systems is a critical step in diagnosis and treatment and helps in educating the affected patients. This paper provides an account of the interplay between the neurological and the musculoskeletal system, how the racial and ethnic variables impact the physiological functioning, and how these processes interact to affect the patient.
Pathophysiologic processes that would account for the patient presenting these symptoms
Conspicuously, the patient has a history of hypertension and smoking that form the basis of his clinical symptoms including left-sided weakness of both the upper and lower limbs and a mild left facial droop. These features suggest a stroke. Hypertension is a well-established cause of stroke through various mechanisms that include acceleration of the arteriosclerotic process in the cerebral arteries thus increasing the risk of stenosis, embolism, and consequently infarction. Furthermore, high intravascular pressure within the cerebral arteries leads to considerable damage to the endothelial lining and alteration of the smooth muscle function (Wajngarten & Silva, 2019). This endothelial damage and altered blood cell-endothelium interaction lead to the formation of local thrombi, ischemia, and cerebral infarction whereas smooth muscle cell degeneration leads to intracerebral hemorrhages.
Smoking on the other hand is associated with an increased risk of developing a stroke. According to Pan et al. (2019) smokers have an increased risk of overall stroke compared with nonsmokers, with a pooled odds ratio of 1.61 (95% CI: 1.34–1.93). Tobacco smoke contains a lot of chemicals that include nicotine, carbon monoxide, and cyanide among many others. These chemicals increase the risk of developing atherosclerosis, platelet aggregation, hypertension, low levels of high-density cholesterol, and high levels of low-density cholesterol. A complex and multifactorial interaction of the abovementioned factors leads to impaired vascular function resulting in both cardiovascular compromise and cerebral embolism and infarction.
Racial/ethnic variables that may impact physiological functioning
Generally, evidence shows that a racial disparity exists in the presentation of this physiological process, affecting more blacks as compared to whites. According to a study by Howard et al. (2019), the black individuals aged between 45 and 64 years had a 3-fold increase of developing stroke as compared with the whites. Black individuals have a higher preponderance of stroke because of their high risk to develop metabolic conditions such as diabetes mellitus, hypertension, obesity, hypercholesterolemia, and smoking (Tong et al., 2021).
How these processes interact to affect the patient
Neurological and musculoskeletal processes work hand in hand. Every movement the body makes requires communication between the brain and the muscles. Consequently, a pathologic process affecting the neurological process can lead to musculoskeletal manifestations such as hemiplegia, muscle spasms, muscle atrophy, and muscle pain among others. For instance, chronic musculoskeletal pain is principally considered a nervous system disorder as a result of nervous system plasticity (George & Bishop, 2018). Similarly, several factors such as smoking, hypertension, aging, and racial variables impact these physiologic systems resulting in an inherent complexity of interactions that affect patient functioning.
Conclusion
Musculoskeletal and neurological systems are correlated. Nurses must assess both systems at a given point for effective diagnosis and treatment. It is also elemental to educate the patient concerning modifiable risk factors such as smoking and hypertension that result in occurrences that limit the functioning of the musculoskeletal and neurological systems.
Also Read:
NURS 6501 Module 5 Assignment: Case Study Analysis
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NURS 6501 Knowledge Check Concepts Of Pediatrics
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NURS 6501 Advanced Pathophysiology Week 1 Discussion
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NURS 6501 Musculoskeletal, metabolic, and multisystem health dysfunctions
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References
George, S. Z., & Bishop, M. D. (2018). Chronic musculoskeletal pain is a nervous system disorder… now what? Physical Therapy, 98(4), 209–213. https://doi.org/10.1093/ptj/pzy002
Howard, V. J., Madsen, T. E., Kleindorfer, D. O., Judd, S. E., Rhodes, J. D., Soliman, E. Z., Kissela, B. M., Safford, M. M., Moy, C. S., McClure, L. A., Howard, G., & Cushman, M. (2019). Sex and race differences in the association of incident ischemic stroke with risk factors. JAMA Neurology, 76(2), 179–186. https://doi.org/10.1001/jamaneurol.2018.3862
Pan, B., Jin, X., Jun, L., Qiu, S., Zheng, Q., & Pan, M. (2019). The relationship between smoking and stroke: A meta-analysis: A meta-analysis. Medicine, 98(12), e14872. https://doi.org/10.1097/MD.0000000000014872
Tong, X., Schieb, L., George, M. G., Gillespie, C., Merritt, R. K., & Yang, Q. (2021). Racial/ethnic and geographic variations in long-term survival among Medicare beneficiaries after acute ischemic stroke. Preventing Chronic Disease, 18(200242), E15. https://doi.org/10.5888/pcd18.200242
Wajngarten, M., & Silva, G. S. (2019). Hypertension and stroke: Update on treatment. European Cardiology, 14(2), 111–115. https://doi.org/10.15420/ecr.2019.11.1
Sample Answer 2 for NURS 6501 Concepts of Neurological and Musculoskeletal Disorders – Part 1
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Question 2
4 out of 4 points
Scenario 1: GoutA 68-year-old obese male presents to the clinic with a 3-day history of fever with chills, and Lt. great toe pain that has gotten progressively worse. Patient states this is the first time that this has happened, and nothing has made it better and walking on his right foot makes it worse. He has tried acetaminophen, but it did not help. He took several ibuprofen tablets last night which did give him a bit of relief. HPI: hypertension treated with Lisinopril/HCTZ . SH: Denies smoking. Drinking: “a fair amount of red wine” every week. General appearance: Ill appearing male who sits with his right foot elevated. PE: remarkable for a temp of 100.2, pulse 106, respirations 20 and BP 158/92. Right great toe (first metatarsal phalangeal [MTP]) noticeably swollen and red. Unable to palpate to assess range of motion due to extreme pain. CBC and Complete metabolic profile revealed WBC 15,000 mm3 and uric acid 9.0 mg/dl. Diagnoses the patient with acute gout. Question:Explain why a patient with gout is more likely to develop renal calculi. |
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In this exercise, you will complete a 10- to 20-essay type question Knowledge Check to gauge your understanding of this module’s content.
Possible topics covered in this Knowledge Check include:
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- Stroke
- Multiple sclerosis
- Transient Ischemic Attack
- Myasthenia gravis
- Headache
- Seizure disorders
- Head injury
- Spinal cord injury
- Inflammatory diseases of the musculoskeletal system
- Osteoporosis
- Osteopenia
- Bursitis
- Tendinitis
- Gout
- Lyme Disease
- Spondylosis
- Fractures
- Parkinson’s
- Alzheimer’s
Three basic bone-formations:
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- Osteoblasts
- Osteocytes
- Osteoclasts
Photo Credit: Getty Images/Science Photo Libra
Complete the Knowledge Check By Day 7 of Week 7
To complete this Knowledge Check:
Module 5 Knowledge Check
Next Week
Week 7: Concepts of Neurological and Musculoskeletal Disorders – Part 1
Anatomists often use the analogy of a house to explain the human body, with skeletal systems, respiratory systems, and circulatory systems represented as a home’s framing structure, ventilation, and piping, respectively. Such analogies further emphasize the point that relationships between systems can result in complications when issues arise in one system.
With hundreds of diseases that can impact the brain, spine, and nerves, neurological disorders represent a complicated array of issues
that present significant health concerns. Disorders such as strokes and Parkinson’s disease not only affect the nervous system, however; they can have secondary impacts in other areas, especially the musculoskeletal system.
This week, you examine fundamental concepts of neurological disorders. You explore common disorders that impact these systems and you apply the key terms and concepts that help communicate the pathophysiological nature of these issues to patients.
Learning Objectives
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Students will:
- Analyze concepts and principles of pathophysiology across the lifespan
Learning Resources
Required Readings (click to expand/reduce)
McCance, K. L. & Huether, S. E. (2019). Pathophysiology: the biologic basis for disease in adults and children (8th ed.). St. Louis, MO: Mosby/Elsevier.
- Chapter 15: Structure and Function of the Neurologic System
- Chapter 16: Pain, Temperature Regulation, Sleep, and Sensory Function (stop at Sleep); Summary Review
- Chapter 17: Alterations in Cognitive Systems, Cerebral Hemodynamics, and Motor Function (start at Acute confusional states and delirium) (stop at Alterations in neuromotor functions); (Parkinson’s Disease); Summary Review
- Chapter 18: Disorders of the Central and Peripheral Nervous Systems and the Neuromuscular Junction (stop at Degenerative disorders of the spine); (start at Cerebrovascular disorders) (stop at Tumors of the central nervous system); Summary Review
- Chapter 44: Structure and Function of the Musculoskeletal System (stop at Components of muscle function); Summary Review
- Chapter 45: Alterations of Musculoskeletal Function (stop at Bone tumors); (start at Disorders of joints); Summary Review
- Chapter 47: Structure, Function, and Disorders of the Integument (section on Lyme Disease)
Required Media (click to expand/reduce)
Module 5 Overview with Dr. Tara Harris
Dr. Tara Harris reviews the structure of Module 5 as well as the expectations for the module. Consider how you will manage your time as you review your media and Learning Resources throughout the module to prepare for your Knowledge Check and your Assignment. (3m)
Note: The approximate length of the media program is 8 minutes.
Note: The approximate length of the media program is 9 minutes.
Online Media from Pathophysiology: The Biologic Basis for Disease in Adults and Children
In addition to this week’s media, it is highly recommended that you access and view the resources included with the course text, Pathophysiology: The Biologic Basis for Disease in Adults and Children. Focus on the videos and animations in Chapters 15, 16, 18, and sections of Chapters 44 and 45 that relate to the neurological and musculoskeletal systems. Refer to the Learning Resources in Week 1 for registration instructions. If you have already registered, you may access the resources at https://evolve.elsevier.com/
Sample Answer 3 for NURS 6501 Concepts of Neurological and Musculoskeletal Disorders – Part 1
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Question 16
Needs Grading
A 22-year-old male is in the Surgical Intensive Care Unit (SICU) following a motor vehicle crash (MVC) where he sustained multiple life-threatening injuries including a torn aorta, ruptured spleen, and bilateral femur fractures. He has had difficulty maintaining his mean arterial pressure (MAP) and has required various vasopressors. He has a triple lumen central venous catheter (CVC) for monitoring his central venous pressure, administration of medications and blood products, as well as total parenteral nutrition. Per hospital protocol, he is receiving an unfractionated heparin 1:1000 flush after administration of each of the triple antibiotics that have been ordered to maintain patency of the lumens. Seven days post injury, the APRN in the SICU is reviewing the patient’s morning labs and notes that his platelet count has dropped precipitously to 50,000 /mm3 from 148,000/mm3 two days ago. The APRN suspects the patient is developing heparin induced thrombocytopenia (HIT).
Question 1 of 2:
What is underlying pathophysiology of heparin induced thrombocytopenia? |
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Question 17
Needs Grading
A 22-year-old male is in the Surgical Intensive Care Unit (SICU) following a motor vehicle crash (MVC) where he sustained multiple life-threatening injuries including a torn aorta, ruptured spleen, and bilateral femur fractures. He has had difficulty maintaining his mean arterial pressure (MAP) and has required various vasopressors. He has a triple lumen central venous catheter (CVC) for monitoring his central venous pressure, administration of medications and blood products, as well as total parenteral nutrition. Per hospital protocol, he is receiving an unfractionated heparin 1:1000 flush after administration of each of the triple antibiotics that have been ordered to maintain patency of the lumens. Seven days post injury, the APRN in the SICU is reviewing the patient’s morning labs and notes that his platelet count has dropped precipitously to 50,000 /mm3 from 148,000/mm3 two days ago. The APRN suspects the patient is developing heparin induced thrombocytopenia (HIT).
Question 2 of 2:
The APRN assesses the patient and notes there is a decreased right posterior tibial pulse with cyanosis of the entire foot. The APRN recognizes this probably represents arterial thrombus formation. How does someone who is receiving heparin develop arterial and venous thrombosis? |
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Sample Answer 4 for NURS 6501 Concepts of Neurological and Musculoskeletal Disorders – Part 1
Alzheimer’s is a common progressive neurological disorder caused by dementia among older adults. The disorder is characterized by mild to moderate cognitive symptoms at first which lead to severe memory loss as the individual ages. Several treatment options have however been provided to help promote the quality of life and reduce suffering among patients diagnosed with this disorder (Tsolaki, 2018). The purpose of this paper is to demonstrate the choice of medication based on pharmacokinetic and pharmacodynamic factors for an elderly patient with Alzheimer’s disease.
Patient Case Study Summary
The patient in the provided case study is a 76-year-old Iranian male who presented to the clinic with strange behaviors. The patient reports symptoms of Alzheimer’s such as confusion, loss of interest in religious activities, and forgetfulness for the past 2 years. Some of the pharmacodynamic and pharmacokinetic factors that might affect the choice of medication for the patient include his male gender, advanced age, and Iranian race, in addition to the mini-mental examination results which reveal moderate dementia (Tan et al., 2018). The patient’s diagnosis of major neurocognitive disorder resulting from Alzheimer’s disease will also be considered.
Treatment Decisions
Based on the above-mentioned pharmacokinetic and pharmacodynamic factors, the best treatment choice for the patient out of the available options was to initiate 1.5 mg Exelon (rivastigmine) twice daily. Studies have reported great effectiveness with the use of rivastigmine as the first-line medication for the management of Alzheimer’s disease, with great tolerance and safety profile (dos Santos et al., 2018). The second intervention was to titrate the dose upwards from 1.5mg to 4.5 mg twice daily as recommended by clinical guidelines, given that the patient displayed limited reemission of symptoms with the use of 1.5 mg rivastigmine for the past 4 weeks. Despite the patient exhibiting great tolerance to the medication, its effectiveness was still limited which led to the final decision to increase the dose to 6mg orally twice daily.
Expected Outcome
Studies show that rivastigmine may take up to 8 to 12 weeks to completely manage the symptoms of dementia. The dose must however be titrated at intervals of 1.5mg after every 2 weeks, after an initial dose of 1.5mg twice daily, to attain the optimum dose for an effective outcome. The patient should however not exceed 6mg twice daily (Nguyen et al., 2021). As such, the patient was expected to display remission of symptoms, within 8 weeks, with minimal mental examination results of less than 10.
Difference Between Expected Outcome and Actual Outcome
The patient responded adequately to the medication as expected. Within the first four weeks, minimal remission of symptoms was experienced with no side effects as expected. The dose was increased which led to better results until the optimum dose was attained with no side effects (Khoury et al., 2018). As such the patient exhibited great tolerance and adherence to the medication with great effectiveness just as expected.
Conclusion
Alzheimer’s disorder is a disabling condition that requires timely treatment to promote the patient’s quality of life. Several treatment options are however available which require keen consideration of the patient’s pharmacokinetic and pharmacodynamic factors when selecting which drug to use for which patient. The 76-year-old Iranian male patient described above displayed great tolerance and adherence to the use of rivastigmine.
References
dos Santos, P., Leide, C., Ozela, P. F., de Fatima de Brito, M., Pinheiro, A. A., Padilha, E. C., … & Izabel, L. (2018). Alzheimer’s disease: a review from the pathophysiology to diagnosis, new perspectives for pharmacological treatment. Current medicinal chemistry, 25(26), 3141-3159. https://doi.org/10.2174/0929867323666161213101126
Khoury, R., Rajamanickam, J., & Grossberg, G. T. (2018). An update on the safety of current therapies for Alzheimer’s disease: focus on rivastigmine. Therapeutic Advances in Drug Safety, 9(3), 171-178. https://doi.org/10.1177/2042098617750555
Nguyen, K., Hoffman, H., Chakkamparambil, B., & Grossberg, G. T. (2021). Evaluation of rivastigmine in Alzheimer’s disease. Neurodegenerative Disease Management, 11(1), 35-48. https://doi.org/10.2217/nmt-2020-0052
Tan, E. C., Hilmer, S. N., Garcia-Ptacek, S., & Bell, J. (2018). Current approaches to the pharmacological treatment of Alzheimer’s disease. Australian Journal of general practice, 47(9), 586-592. https://search.informit.org/doi/10.3316/informit.849432651288623
Tsolaki, M. (2018). An old and new challenge for the treatment of Alzheimer’s disease. Journal of Neurology and Neuroscience, 09. https://doi.org/10.21767/2171-6625-c3-013