NURS 6051 Knowledge Check: Cardiovascular And Respiratory Disorders
Walden University NURS 6051 Knowledge Check: Cardiovascular And Respiratory Disorders– Step-By-Step Guide
This guide will demonstrate how to complete the Walden University NURS 6051 Knowledge Check: Cardiovascular And Respiratory Disorders assignment based on general principles of academic writing. Here, we will show you the A, B, Cs of completing an academic paper, irrespective of the instructions. After guiding you through what to do, the guide will leave one or two sample essays at the end to highlight the various sections discussed below.
How to Research and Prepare for NURS 6051 Knowledge Check: Cardiovascular And Respiratory Disorders
Whether one passes or fails an academic assignment such as the Walden University NURS 6051 Knowledge Check: Cardiovascular And Respiratory Disorders depends on the preparation done beforehand. The first thing to do once you receive an assignment is to quickly skim through the requirements. Once that is done, start going through the instructions one by one to clearly understand what the instructor wants. The most important thing here is to understand the required format—whether it is APA, MLA, Chicago, etc.
After understanding the requirements of the paper, the next phase is to gather relevant materials. The first place to start the research process is the weekly resources. Go through the resources provided in the instructions to determine which ones fit the assignment. After reviewing the provided resources, use the university library to search for additional resources. After gathering sufficient and necessary resources, you are now ready to start drafting your paper.
How to Write the Introduction for NURS 6051 Knowledge Check: Cardiovascular And Respiratory Disorders
The introduction for the Walden University NURS 6051 Knowledge Check: Cardiovascular And Respiratory Disorders is where you tell the instructor what your paper will encompass. In three to four statements, highlight the important points that will form the basis of your paper. Here, you can include statistics to show the importance of the topic you will be discussing. At the end of the introduction, write a clear purpose statement outlining what exactly will be contained in the paper. This statement will start with “The purpose of this paper…” and then proceed to outline the various sections of the instructions.
How to Write the Body for NURS 6051 Knowledge Check: Cardiovascular And Respiratory Disorders
After the introduction, move into the main part of the NURS 6051 Knowledge Check: Cardiovascular And Respiratory Disorders assignment, which is the body. Given that the paper you will be writing is not experimental, the way you organize the headings and subheadings of your paper is critically important. In some cases, you might have to use more subheadings to properly organize the assignment. The organization will depend on the rubric provided. Carefully examine the rubric, as it will contain all the detailed requirements of the assignment. Sometimes, the rubric will have information that the normal instructions lack.
Another important factor to consider at this point is how to do citations. In-text citations are fundamental as they support the arguments and points you make in the paper. At this point, the resources gathered at the beginning will come in handy. Integrating the ideas of the authors with your own will ensure that you produce a comprehensive paper. Also, follow the given citation format. In most cases, APA 7 is the preferred format for nursing assignments.
How to Write the Conclusion for NURS 6051 Knowledge Check: Cardiovascular And Respiratory Disorders
After completing the main sections, write the conclusion of your paper. The conclusion is a summary of the main points you made in your paper. However, you need to rewrite the points and not simply copy and paste them. By restating the points from each subheading, you will provide a nuanced overview of the assignment to the reader.
How to Format the References List for NURS 6051 Knowledge Check: Cardiovascular And Respiratory Disorders
The very last part of your paper involves listing the sources used in your paper. These sources should be listed in alphabetical order and double-spaced. Additionally, use a hanging indent for each source that appears in this list. Lastly, only the sources cited within the body of the paper should appear here.
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Sample Answer for NURS 6051 Knowledge Check: Cardiovascular And Respiratory Disorders
Scenario 1: Myocardial Infarction
CC: “I woke up this morning at 6 a.m. with numbness in my left arm and pain in my chest. It feels tight right here (mid-sternal).” “My dad had a heart attack when he was 56-years-old and I am scared because I am 56-years-old.”
HPI: Patient is a 56-year-old Caucasian male who presents to Express Hospital Emergency Department with a chief complaint of chest pain that radiates down his left arm. He states this started this morning and has been getting worse, pointing to the mid-sternal area, “it feels like an elephant is sitting on my chest and having a hard time breathing”. He rates the pain as 9/10. Nothing has made the pain better or worse. He denies any previous episode of chest pain. Denies nausea, or lightheadedness. Nitroglycerin 0.4 mg tablet sublingual x 1 which decreased pain to 7/10.
Lipid panel reveals Total Cholesterol 424 mg/dl, high density lipoprotein (HDL) 26 mg/dl, Low Density Lipoprotein (LDL) 166 mg/dl, Triglycerides 702 mg/dl, Very Low-Density Lipoprotein (VLDL) 64 mg/dl
His diagnosis is an acute inferior wall myocardial infarction.
Question:
Which cholesterol is considered the “good” cholesterol and what does it do?
High-density lipoprotein (HDL) is also called the “good” cholesterol, while low-density lipoprotein (LDL) is called “bad” cholesterol. HDL absorbs cholesterol in the blood and transports it back to the liver. The liver then flushes the cholesterol from the body. High levels of HDL cholesterol lower the risk of heart disease and stroke. HDL has a diverse protein and lipid composition, contributing to its atheroprotective function (Jomard & Osto, 2020). In the vessel wall, HDL undergoes transcytosis through endothelial cells into the sub-endothelial space, where it efflux cholesterol from foam cells, preventing plaque formation. In addition, HDLs have other beneficial properties, like nitric oxide production stimulation, anti-oxidant capacity, anti-inflammatory, and anti-apoptotic actions.
Scenario 1: Myocardial Infarction
CC: “I woke up this morning at 6 a.m. with numbness in my left arm and pain in my chest. It feels tight right here (mid-sternal).” “My dad had a heart attack when he was 56-years-old and I am scared because I am 56-years-old.”
HPI: Patient is a 56-year-old Caucasian male who presents to Express Hospital Emergency Department with a chief complaint of chest pain that radiates down his left arm. He states this started this morning and has been getting worse, pointing to the mid-sternal area, “it feels like an elephant is sitting on my chest and having a hard time breathing”. He rates the pain as 9/10. Nothing has made the pain better or worse. He denies any previous episode of chest pain. Denies nausea, or lightheadedness. Nitroglycerin 0.4 mg tablet sublingual x 1 which decreased pain to 7/10.
Lipid panel reveals Total Cholesterol 424 mg/dl, high density lipoprotein (HDL) 26 mg/dl, Low Density Lipoprotein (LDL) 166 mg/dl, Triglycerides 702 mg/dl, Very Low-Density Lipoprotein (VLDL) 64 mg/dl
His diagnosis is an acute inferior wall myocardial infarction.
Question:
- How does inflammation contribute to the development of atherosclerosis?
Atherosclerosis is a chronic inflammatory condition with an autoimmune component. It is accompanied by a chronic, low-grade inflammatory response that attracts cells of the innate and adaptive immune systems into the atherosclerotic plaque (Wolf & Ley, 2019). The autoimmune response is clinically best documented by antibodies against LDL and other atherosclerosis antigens. The continued development of atherosclerosis involves an inflammatory response, which begins with injury to the vascular endothelium. Inflammation has multiple effects on the arterial wall, including attracting inflammatory cells like macrophages (Jebari-Benslaiman et al., 2022). The macrophages infiltrate the injured vascular endothelium and ingest lipids, making them foam cells. Activated macrophages release biochemical substances that can further damage the endothelium, attracting platelets and initiating clotting.
Scenario 2: Pleural Friction Rub
A 35-year-old female with a positive history of systemic lupus erythematosus (SLE) presents to the Emergency Room (ER) with complaints of sharp retrosternal chest pain that worsens with deep breathing or lying down. She reports a 5-day history of low-grade fever, listlessness and says she feels like she had the flu. Physical exam reveals tachycardia and a pleural friction rub. She was diagnosed with acute pericarditis.
Question:
- Because of the result of a pleural friction rub, what does the APRN recognize?
Physical findings in a pleural friction rub include an adventitious breath sound on chest auscultation, which is heard as a harsh, grunting sound during systole and diastole. When examining the patient with a friction rub, the APRN will identify that SLE caused an inflammation of the pericardium resulting in pericarditis (Dein et al., 2019). When the patient was in active lupus, antigen-antibody complexes formed and mediated inflammation of the pericardium. Therefore, the APRN will recognize that the client’s symptoms are caused by pericarditis, which is characterized by fever, dyspnea, tachycardia, and faint heart sounds. Pericardial rub is a common finding on physical exam.
Scenario 4: Deep Venous Thrombosis (DVT)
A 81-year-old obese female patient who 48 hours post-op left total hip replacement. The patient has had severe nausea and vomiting and has been unable to go to physical therapy. Her mucus membranes are dry. The patient says the skin on her left leg is too tight. Exam reveals a swollen, tense, and red colored calf. The patient has a duplex ultrasound which reveals the presence of a deep venous thrombosis (DVT).
Question:
- Given the history of the patient explain what contributed to the development of a deep venous thrombosis (DVT)
The patient’s DVT was caused by venous stasis, activation of the coagulation pathway, and vein damage. The patient was at risk of developing a thrombus due to being obese. The patient likely developed Venous stasis following vein damage during the total hip replacement. Immobility of the left leg following surgery and failure to attend physical therapy caused venous stasis contributing to DVT (Chindamo & Marques, 2019). The venous stasis caused an increased viscosity resulting in the formation of microthrombi, which are not cleared by fluid movement when there is high blood viscosity. This resulted in the formation of a blood clot from the microthrombi, which interrupted blood flow in the vein.
Scenario 5: COPD
A 66-year-old female with a 50 pack/year history of cigarette smoking had a CT scan and was diagnosed with emphysema. He asks if this means he has chronic obstructive pulmonary disease (COPD).
Question:
- There is a clear relationship between emphysema and COPD, explain the pathophysiology of emphysema and the relationship to COPD.
Emphysema is characterized by two significant changes: loss of lung elasticity and hyperinflation of the lung. It occurs when protein-degrading enzymes called proteases are at higher-than-normal levels. They damage the alveoli and the small airways by breaking down elastin (Rodrigues et al., 2021). High protease levels make the alveolar sacs lose their elasticity, and the small airways collapse or narrow. Some alveoli get destroyed, and others become large and flaccid with reduced area for effective gas exchange. An increased amount of air is trapped in the lungs due to loss of elastic recoil in the alveolar walls, overstretching and enlargement of the alveoli into air-filled spaces, and collapse of small airways (Rodrigues et al., 2021). Emphysema is linked to COPD since oxygenation is affected by the loss of alveolar tissue and the increased work of breathing. Usually, inhalation begins before exhalation is completed, causing an uncoordinated breathing pattern.
References
Chindamo, M. C., & Marques, M. A. (2019). Role of ambulation to prevent venous thromboembolism in medical patients: where do we stand?. Jornal Vascular Brasileiro, 18. https://doi.org/10.1590/1677-5449.180107
Dein, E., Douglas, H., Petri, M., Law, G., & Timlin, H. (2019). Pericarditis in Lupus. Cureus, 11(3), e4166. https://doi.org/10.7759/cureus.4166
Jebari-Benslaiman, S., Galicia-García, U., Larrea-Sebal, A., Olaetxea, J. R., Alloza, I., Vandenbroeck, K., Benito-Vicente, A., & Martín, C. (2022). Pathophysiology of Atherosclerosis. International journal of molecular sciences, 23(6), 3346. https://doi.org/10.3390/ijms23063346
Jomard, A., & Osto, E. (2020). High density lipoproteins: metabolism, function, and therapeutic potential. Front Cardiovasc Med. 2020; 7: 39. https://doi.org/10.3389/fcvm.2020.00039
Rodrigues, S. O., Cunha, C. M. C. D., Soares, G. M. V., Silva, P. L., Silva, A. R., & Gonçalves-de-Albuquerque, C. F. (2021). Mechanisms, Pathophysiology and Currently Proposed Treatments of Chronic Obstructive Pulmonary Disease. Pharmaceuticals (Basel, Switzerland), 14(10), 979. https://doi.org/10.3390/ph14100979
Wolf, D., & Ley, K. (2019). Immunity and inflammation in atherosclerosis. Circulation research, 124(2), 315-327.https://doi.org/10.1161/CIRCRESAHA.118.313591
Sample Answer 2 for NURS 6051 Knowledge Check: Cardiovascular And Respiratory Disorders
Question 1
2 out of 2 points
CC: “I have been having terrible chest and arm pain for the past 2 hours and I think I am having a heart attack.”
HPI: Mr. Hammond is a 57-year-old African American male who presents to the Emergency Department with a chief complaint of chest pain that radiates down his left arm. He states that he started having pain several hours ago and says the pain “it feels like an elephant is sitting on my chest”. He rates the pain as 8/10. Nothing has made the pain better or worse. He denies any previous episode of chest pain. Denies nausea, dyspnea, or lightheadedness. He was given 0.4 mg nitroglycerine tablet sublingual x 1 which decreased, but not stopped the pain. Lipid panel reveals Total Cholesterol 324 mg/dl, high density lipoprotein (HDL) 31 mg/dl, Low Density Lipoprotein (LDL) 122 mg/dl, Triglycerides 402 mg/dl, Very Low-Density Lipoprotein (VLDL) 54 mg/dl His diagnosis is an acute inferior wall myocardial infarction. Musculoskeletal, metabolic, and multisystem health dysfunctions represent one of the most challenging health conditions that may have a complex challenge to the affected patient (Beefcroft & Hough, 2016). Symptoms of these conditions may range in severity. Musculoskeletal complications can affect the muscle, bone, tendons and joints. A major symptom that patients encounter is pain. Conditions may range from the ones that are sudden and short-lived to the ones that are lifelong and are associated with disability as well as ongoing pain. Among the notable changes that a patient with this condition may experience include persistent pain as well as mobility limitations. In this, the patients who may suffer from the long-term condition may experience severe pain that may be related to the wearing out of the tendons. For some patients with complex conditions, they may experience joint deformity that may be a long term condition, and it may be relatively challenging to treat it. When these abnormal changes occur, the involved patient may find it hard to handle some of the changes in weight, thus affecting some parts of the body (Black, 2016). Although musculoskeletal conditions may arise in many forms as well as a result of different factors, there are many ways that the situation may represent itself. The identification of specific symptoms of the disorder plays an essential role in the development of an approach to assist in the management of the stated condition. 1 of 2 Questions:Why is HDL considered the “good” cholesterol? |
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Question 2
3 out of 3 points
CC: “I have been having terrible chest and arm pain for the past 2 hours and I think I am having a heart attack.”
HPI: Mr. Hammond is a 57-year-old African American male who presents to the Emergency Department with a chief complaint of chest pain that radiates down his left arm. He states that he started having pain several hours ago and says the pain “it feels like an elephant is sitting on my chest”. He rates the pain as 8/10. Nothing has made the pain better or worse. He denies any previous episode of chest pain. Denies nausea, dyspnea, or lightheadedness. He was given 0.4 mg nitroglycerine tablet sublingual x 1 which decreased, but not stopped the pain. Lipid panel reveals Total Cholesterol 324 mg/dl, high density lipoprotein (HDL) 31 mg/dl, Low Density Lipoprotein (LDL) 122 mg/dl, Triglycerides 402 mg/dl, Very Low-Density Lipoprotein (VLDL) 54 mg/dl His diagnosis is an acute inferior wall myocardial infarction. 2 of 2 Questions:Explain the role inflammation has in the development of atherosclerosis. |
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Question 3
1 out of 1 points
A 45-year-old woman with a history of systemic lupus erythematosus (SLE) presents to the Emergency Room (ER) with complaints of sharp retrosternal chest pain that worsens with deep breathing or lying down. She reports a 3-day history of low-grade fever, listlessness and says she feels like she had the flu. Physical exam reveals tachycardia and a pleural friction rub. She was diagnosed with acute pericarditis.
Question:
What does the Advanced Practice Registered Nurse (APRN) recognize as the result of the pleural friction rub? |
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Question 4
1 out of 1 points
A 15-year-old adolescent male comes to the clinic with his parents with a chief complaint of fever, nausea, vomiting, poorly localized abdominal pain, arthralgias, and “swollen lymph nodes”. States he has felt “lousy” for a couple weeks. The fevers have been as high as 102 F. His parents thought he had the flu and took him to an Urgent Care Center. He was given Tamiflu® and sent home. He says the Tamiflu didn’t seem to work. States had a slight sore throat a couple weeks ago and attributed it to the flu. Physical exam revealed thin young man who appears to be uncomfortable but not acutely ill. Posterior pharynx reddened and tonsils 3+ without exudate. + anterior and posterior cervical lymphadenopathy. Tachycardic and a new onset 2/6 high-pitched, crescendo-decrescendo systolic ejection murmur auscultated at the left sternal border. Rapid strep +. The patient was diagnosed with acute rheumatic heart disease (RHD)
ALSO READ: Assignment 2: Digital Clinical Experience (DCE): Health History Assessment NURS 6512N-32Question:
Explain how a positive strep test has caused the patient’s symptoms. |
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Question 5
1 out of 1 points
The APRN sees a 74-year-old obese female patient who is 2 days post-op after undergoing left total hip replacement. The patient has had severe post op nausea and vomiting and has been unable to go to physical therapy. Her mucus membranes are dry. The patient says she feels like the skin on her left leg is too tight. Exam reveals a swollen, tense, and red colored calf. The patient has a duplex ultrasound which reveals the presence of a deep venous thrombosis (DVT).
Question:Describe the factors that could have contributed to the development of a DVT in this patient explain how each of the factors could cause DVT. |
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Question 6
1 out of 1 points
A 45-year-old woman is 10 days status post partial small bowel resection for Crohn Disease and has been recuperating at home. She suddenly develops severe shortness of breath, becomes weak, and her blood pressure drops to 80/40 mmHg (previous readings ~130/80s mmHg). The pulse oximetry is 89% on room air. The APRN suspects the patient experienced a massive pulmonary embolus.
Question:
Explain why a large pulmonary embolus interferes with oxygenation. |
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Question 7
1 out of 1 points
A 45-year-old woman is 10 days status post partial small bowel resection for Crohn Disease and has been recuperating at home. She suddenly develops severe shortness of breath, becomes weak, and her blood pressure drops to 80/40 mmHg (previous readings ~130/80s mmHg). The pulse oximetry is 89% on room air. While waiting for the Emergency Medical Service (EMS) to arrive, the APRN places EKG leads and the EKG demonstrates right ventricular strain.
Question:
Explain why a large pulmonary embolism causes right ventricular strain. |
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Question 8
2 out of 2 points
A 12-year-old girl is brought to the Emergency Room (ER) by her mother with complaints of shortness of breath, wheezing, tachypnea, tachycardia, and a non-productive cough. The mother states they had just come from a fall festival where the entire family enjoyed a hayride. The symptoms began shortly after they left the festival but got better a couple hours after they returned home. The symptoms began again about 6 hours later and seem to be worse. The mother states there is no history of allergies or frequent respiratory infections. The child is up to date on all vaccinations. The child was diagnosed with asthma. The nurse practitioner explained to the mother that her child was exhibiting symptoms of asthma, and probably had an early asthmatic response and a late asthmatic response.
Question 1 of 2:
Explain early asthmatic responses and the cells responsible for the responses. |
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Question 9
1.8 out of 2 points
A 12-year-old girl is brought to the Emergency Room (ER) by her mother with complaints of shortness of breath, wheezing, tachypnea, tachycardia, and a non-productive cough. The mother states they had just come from a fall festival where the entire family enjoyed a hayride. The symptoms began shortly after they left the festival but got better a couple hours after they returned home. The symptoms began again about 6 hours later and seem to be worse. The mother states there is no history of allergies or frequent respiratory infections. The child is up to date on all vaccinations. The child was diagnosed with asthma. The nurse practitioner explained to the mother that her child was exhibiting symptoms of asthma, and probably had an early asthmatic response and a late asthmatic response.
Question 2 of 2:Explain late asthmatic responses and the cells responsible for the responses. |
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Question 10
2 out of 2 points
A 64-year-old man with a 40 pack/year history of cigarette smoking has been diagnosed with emphysema. He asks the APRN if this means he has COPD.
Question 1 of 2:Explain the pathophysiology of emphysema and how it relates to COPD. |
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Question 11
2 out of 2 points
A 64-year-old man with a 40 pack/year history of cigarette smoking has been diagnosed with emphysema. He asks the APRN if this means he has COPD.
Question 2 of 2:Explain the pathophysiology of chronic bronchitis and how it relates to COPD. |
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Question 12
1 out of 1 points
Mr. Jones is a 78-year-old gentleman who presents to the clinic with a chief complaint of fever, chills and cough. He also reports some dyspnea. He has a history of right sided CVA, COPD, dyslipidemia, and HTN. Current medications include atorvastatin 40 mg po qhs, lisinopril, and fluticasone/salmeterol. He reports more use of his albuterol rescue inhaler.
Vital signs Temp 101.8 F, pulse 108, respirations 21. PaO2 on room air 86% and on O2 4 L nasal canula 94%. CMP WNL, WBC 18.4. Physical exam reveals thin, anxious gentleman with mild hemiparesis on left side due to CVA. HEENT WNL except for diminished gag reflex and uneven elevation of the uvula, CV-HR 108 RRR without murmurs, rubs, or click, no bruits. Resp-coarse rhonchi throughout lung fields. CXR reveals consolidation in right lower lobe. He was diagnosed with community acquired pneumonia (CAP). Question:
Patient was hypoxic as evidenced by the low PaO2. Explain the pathologic processes that caused this patient’s hypoxemia. |
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Question 13
1 out of 1 points
A 64-year-old woman with moderately severe COPD comes to the pulmonary clinic for her quarterly checkup. The APRN reviewing the chart notes that the patient has lost 5% of her body weight since her last visit. The APRN questions the patient and patient admits to not having much of an appetite and she also admits to missing some meals because it “takes too much work” to cook and consume dinner.
Question:
The APRN recognizes that COPD has a deleterious effect on patients. Explain why patients with COPD are at risk for malnutrition. |
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Review Test Submission: Module 2 Knowledge Check
The circulatory system and the respiratory system are powerful partners in health. While they work closely together in good health, a disease or disorder that manifests in one can have a significant impact on both, hampering the pair’s ability to collaborate.
Cardiovascular and respiratory disease and disorders are among the most common reasons for hospital visits, and among the leading causes of fatality. Heart disease and pneumonias are among the most familiar, but a wide variety of issues can impact physiological functioning of one or both systems.
This week, you examine fundamental concepts of cardiovascular and respiratory disorders. You explore common diseases and disorders that impact these systems, and you apply the key terms and concepts that help communicate the pathophysiological nature of these issues to patients.
Click here to ORDER NOW FOR AN ORIGINAL PAPER ASSIGNMENT ON NURS 6051 Knowledge Check: Cardiovascular and Respiratory Disorders
Learning Objectives
Students will:
- Analyze concepts and principles of pathophysiology across the lifespan
Learning Resources
Cardiovascular Respiratory Disorders – Week 3 (16m)
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NURS 6051 Advocating for the Nursing Role in Program Design and Implementation
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NURS 6051 promotion of safety, quality, and efficiency
NURS 6051 Software Development Life Cycle (SDLC)
NURS 6051 Discussion mental health disorders that require a long-term treatment follow-up
NURS 6051 Musculoskeletal, metabolic, and multisystem health dysfunctions
Knowledge Check: Cardiovascular And Respiratory Disorders
In this exercise, you will complete a 10- to 20-essay type question Knowledge Check to gauge your understanding of this module’s content.
Possible topics covered in this Knowledge Check include:
- myocardial infarction
- endocarditis
- myocarditis
- valvular disorders
- lipid panels
- coagulation
- clotting cascade
- deep vein thrombosis
- hypertension
- heart failure
- COPD
- asthma
- pneumonias
Photo Credit: Getty Images/Science Photo Library RF
Complete the Knowledge Check by Day 7 of Week 3
To complete this Knowledge Check:
Module 2 Knowledge Check
What’s Coming Up In Week 4?
Photo Credit: [BrianAJackson]/[iStock / Getty Images Plus]/Getty Images
Next week, you will examine the alterations in the cardiovascular and respiratory systems and the resultant disease processes through case study analysis. You will also consider patient characteristics, including racial and ethnic variables, which may impact altered physiology.
Next Week
To go to the next week:
Week 4
Question 1
1 out of 1 points
Correct
Sympathetic stimulation causes airways to:
Selected Answer:
Correct
dilate
Answers:
Correct
dilate
constrict
collapse
trap air
Question 2
1 out of 1 points
Correct
Sympathetic nerves to the heart releases what the neurotransmitter?
Selected Answer:
Correct
norepinephrine
Answers:
serotonin
epinephrine
Correct
norepinephrine
acetylcholine
Question 3
1 out of 1 points
Correct
Norepinephrine action on a1-adrenergic receptors causes ________________________.
Selected Answer:
Correct
vasoconstriction
Answers:
Correct
vasoconstriction
vasodilation
bradycardia
hypotension
Question 4
1 out of 1 points
Correct
A patient that is hyperventilating will have a decreased:
Selected Answer:
Correct
PaCO2
Answers:
saturation
Pa02
Correct
PaCO2
minute volume
Question 5
1 out of 1 points
Correct
A person who has pulmonary edema will exhibit which symptoms?
Selected Answer:
Correct
dullness to percussion over the lung bases, inspiratory crackles, and pink frothy sputum
Answers:
resonance to percussion over the lung bases, inspiratory wheezing, foul smelling sputum
Correct
dullness to percussion over the lung bases, inspiratory crackles, and pink frothy sputum
resonance to percussion over the lung bases, inspiratory wheezing, and pink frothy sputum
dullness to percussion over the lung bases, inspiratory wheezing, foul smelling sputum
Question 6
1 out of 1 points
Correct
Stimulation of the carina often causes:
Selected Answer:
Correct
coughing
Answers:
inhalation
Correct
coughing
gagging
swallowing
Question 7
1 out of 1 points
Correct
As a person ages, what type of changes occur in the myocardium and arterial walls?
Selected Answer:
Correct
stiffening
Answers:
dilation
Correct
stiffening
atrophy
shrink
Question 8
1 out of 1 points
Correct
Parasympathetic stimulation causes airways to:
Selected Answer:
Correct
constrict
Answers:
dilate
Correct
constrict
collapse
trap air
Question 9
1 out of 1 points
Correct
Parasympathetic nerves to the heart releases what the neurotransmitter?
Selected Answer:
Correct
acetylcholine
Answers:
serotonin
epinephrine
norepinephrine
Correct
acetylcholine
Question 10
1 out of 1 points
Correct
Inflammatory mediators released during an acute asthma episode cause:
Selected Answer:
Correct
inflammation, hypersecretion of mucous, and bronchial smooth muscle constriction
Answers:
Correct
inflammation, hypersecretion of mucous, and bronchial smooth muscle constriction
inflammation, bleeding, and bronchial smooth muscle constriction
bronchial smooth muscle dilation, alveolar collapse, and retained PaCO2
bronchial smooth muscle dilation, inflammation, and thick mucous
Question 11
1 out of 1 points
Correct
The presence of pus in the pleural cavity is a(n):
Selected Answer:
Correct
Empyema
Answers:
Correct
Empyema
Atelectasis
Aspiration
Hemoptysis
Question 12
1 out of 1 points
Correct
A person with a respiratory rate of 12 breaths per minute and a minute volume of 6.0 L/minute has a tidal volume of :
Selected Answer:
Correct
500 ml
Answers:
720 ml
600 ml
1000 ml
Correct
500 ml
Question 13
1 out of 1 points
Correct
Ischemic pain in the lower extremities that occurs while walking but disappears when resting is a description of which condition?
Selected Answer:
Correct
Intermittent claudication
Answers:
Pericarditis
Varicose veins
Correct
Intermittent claudication
Thromboangiitis obliterans
Question 14
1 out of 1 points
Correct
Clinical manifestations of bronchiolitis include:
Selected Answer:
Correct
tachypnea, non-productive cough, and low grade fever
Answers:
hypoxia, use of accessory muscles, and high grade fever
tachypnea, use of accessory muscles, and high grade fever
tachypnea, productive cough, and high grade fever
Correct
tachypnea, non-productive cough, and low grade fever
Question 15
1 out of 1 points
Correct
Binding of ATP to myosin that enables myocardial contraction requires which electrolyte?
Selected Answer:
Correct
calcium
Answers:
Correct
calcium
magnesium
sodium
potassium
Question 16
1 out of 1 points
Correct
How much oxygen does the myocardium extract from the coronary arteries?
Selected Answer:
Correct
70%
Answers:
40%
50%
60%
Correct
70%
Question 17
1 out of 1 points
Correct
The right side of the heart is a high-pressure system
Selected Answer:
Correct
False
Answers:
True
Correct
False
Question 18
1 out of 1 points
Correct
Collapse of alveoli is a(n):
Selected Answer:
Correct
Atelectasis
Answers:
Empyema
Aspiration
Correct
Atelectasis
Hemoptysis
Question 19
1 out of 1 points
Correct
The tunica media is the middle layer of blood vessels and is composed of what type of tissue?
Selected Answer:
Correct
smooth muscle and elastic fibers
Answers:
endothelium and elastic fibers
endothelium and connective tissue
Correct
smooth muscle and elastic fibers
smooth muscle and connective tissue
Question 20
1 out of 1 points
Correct
Prinzmetal angina is caused by:
Selected Answer:
Correct
vasospasm of the coronary artery
Answers:
obstruction of a coronary artery
Correct
vasospasm of the coronary artery
thrombus within the coronary artery
dissection of the coronary artery
Sample Answer 3 for NURS 6051 Knowledge Check: Cardiovascular And Respiratory Disorders
Scenario 4: Deep Venous Thrombosis (DVT)A 81-year-old obese female patient who 48 hours post-op left total hip replacement. The patient has had severe nausea and vomiting and has been unable to go to physical therapy. Her mucus membranes are dry. The patient says the skin on her left leg is too tight. Exam reveals a swollen, tense, and red colored calf. The patient has a duplex ultrasound which reveals the presence of a deep venous thrombosis (DVT). Question:1.Given the history of the patient explain what contributed to the development of a deep venous thrombosis (DVT) |
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· Question 5
4 out of 4 points
Scenario 5: COPDA 66-year-old female with a 50 pack/year history of cigarette smoking had a CT scan and was diagnosed with emphysema. He asks if this means he has chronic obstructive pulmonary disease (COPD). Question:1.There is a clear relationship between emphysema and COPD, explain the pathophysiology of emphysema and the relationship to COPD. |
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Sample Answer 4 for NURS 6051 Knowledge Check: Cardiovascular And Respiratory Disorders
Question 1
2 out of 2 points
CC: “I have been having terrible chest and arm pain for the past 2 hours and I think I am having a heart attack.”
HPI: Mr. Hammond is a 57-year-old African American male who presents to the Emergency Department with a chief complaint of chest pain that radiates down his left arm. He states that he started having pain several hours ago and says the pain “it feels like an elephant is sitting on my chest”. He rates the pain as 8/10. Nothing has made the pain better or worse. He denies any previous episode of chest pain. Denies nausea, dyspnea, or lightheadedness. He was given 0.4 mg nitroglycerine tablet sublingual x 1 which decreased, but not stopped the pain. Lipid panel reveals Total Cholesterol 324 mg/dl, high density lipoprotein (HDL) 31 mg/dl, Low Density Lipoprotein (LDL) 122 mg/dl, Triglycerides 402 mg/dl, Very Low-Density Lipoprotein (VLDL) 54 mg/dl His diagnosis is an acute inferior wall myocardial infarction. 1 of 2 Questions: Why is HDL considered the “good” cholesterol? |
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Question 2
3 out of 3 points
CC: “I have been having terrible chest and arm pain for the past 2 hours and I think I am having a heart attack.”
HPI: Mr. Hammond is a 57-year-old African American male who presents to the Emergency Department with a chief complaint of chest pain that radiates down his left arm. He states that he started having pain several hours ago and says the pain “it feels like an elephant is sitting on my chest”. He rates the pain as 8/10. Nothing has made the pain better or worse. He denies any previous episode of chest pain. Denies nausea, dyspnea, or lightheadedness. He was given 0.4 mg nitroglycerine tablet sublingual x 1 which decreased, but not stopped the pain. Lipid panel reveals Total Cholesterol 324 mg/dl, high density lipoprotein (HDL) 31 mg/dl, Low Density Lipoprotein (LDL) 122 mg/dl, Triglycerides 402 mg/dl, Very Low-Density Lipoprotein (VLDL) 54 mg/dl His diagnosis is an acute inferior wall myocardial infarction. 2 of 2 Questions: Explain the role inflammation has in the development of atherosclerosis. |
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